Release of carbon disulfide is a contributing mechanism in the axonopathy produced by N,N-diethyldithiocarbamate.

Johnson DJ, Graham DG, Amarnath V, Amarnath K, Valentine WM
Toxicol Appl Pharmacol. 1998 148 (2): 288-96

PMID: 9473537 · DOI:10.1006/taap.1997.8344

MeSH Terms (15)

Animals Axons Carbon Disulfide Chelating Agents Cross-Linking Reagents Ditiocarb Erythrocytes Hemoglobins Male Neurofilament Proteins Rats Rats, Sprague-Dawley Spectrin Spinal Cord Tibial Nerve

Connections (1)

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