Treatment-Induced Tumor Cell Apoptosis and Secondary Necrosis Drive Tumor Progression in the Residual Tumor Microenvironment through MerTK and IDO1.

Werfel TA, Elion DL, Rahman B, Hicks DJ, Sanchez V, Gonzales-Ericsson PI, Nixon MJ, James JL, Balko JM, Scherle PA, Koblish HK, Cook RS
Cancer Res. 2019 79 (1): 171-182

PMID: 30413412 · DOI:10.1158/0008-5472.CAN-18-1106

MeSH Terms (17)

Animals Antineoplastic Agents Apoptosis c-Mer Tyrosine Kinase Female Indoleamine-Pyrrole 2,3,-Dioxygenase Inflammation Lapatinib Lung Neoplasms Macrophages Mammary Neoplasms, Experimental Mice Necrosis Phagocytosis Receptor, ErbB-2 T-Lymphocytes, Regulatory Tumor Microenvironment

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