Type 2 diabetes and congenital hyperinsulinism cause DNA double-strand breaks and p53 activity in β cells.

Tornovsky-Babeay S, Dadon D, Ziv O, Tzipilevich E, Kadosh T, Schyr-Ben Haroush R, Hija A, Stolovich-Rain M, Furth-Lavi J, Granot Z, Porat S, Philipson LH, Herold KC, Bhatti TR, Stanley C, Ashcroft FM, In't Veld P, Saada A, Magnuson MA, Glaser B, Dor Y
Cell Metab. 2014 19 (1): 109-21

PMID: 24332968 · DOI:10.1016/j.cmet.2013.11.007

MeSH Terms (21)

Animals Biomarkers Calcineurin Cell Death Cell Proliferation Congenital Hyperinsulinism Diabetes Mellitus, Type 2 Disease Models, Animal DNA Breaks, Double-Stranded Enzyme Activation Enzyme Induction Fasting Glucagon-Like Peptide 1 Glucokinase Glucose Humans Insulin-Secreting Cells Membrane Potentials Mice Transgenes Tumor Suppressor Protein p53

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