Antioxidant supplementation ameliorates molecular deficits in Smith-Lemli-Opitz syndrome.

Korade Z, Xu L, Harrison FE, Ahsen R, Hart SE, Folkes OM, Mirnics K, Porter NA
Biol Psychiatry. 2014 75 (3): 215-22

PMID: 23896203 · PMCID: PMC3874268 · DOI:10.1016/j.biopsych.2013.06.013

MeSH Terms (19)

alpha-Tocopherol Animals Animals, Newborn Antioxidants Ascorbic Acid Brain Cell Line, Transformed Disease Models, Animal Female Fibroblasts Gene Expression Regulation Humans Liver Male Mice Mice, Transgenic Oxidoreductases Acting on CH-CH Group Donors Smith-Lemli-Opitz Syndrome Ubiquinone

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