Aprotinin exacerbates left ventricular dysfunction after ischemia/reperfusion in mice lacking tumor necrosis factor receptor I.

Sabbagh MJ, Looper JM, Zavadzkas JA, Stroud RE, Ford RL, Rivers WT, Koval CN, McEvoy MD, Reeves ST, Spinale FG
J Cardiovasc Pharmacol. 2008 52 (4): 355-62

PMID: 18841074 · PMCID: PMC2637802 · DOI:10.1097/FJC.0b013e3181893659

MeSH Terms (13)

Animals Aprotinin Mice Mice, Inbred C57BL Mice, Transgenic Models, Animal Myocardium Random Allocation Receptors, Tumor Necrosis Factor, Type I Reperfusion Injury Serine Proteinase Inhibitors Tumor Necrosis Factor-alpha Ventricular Dysfunction, Left

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