Elimination of GD3 synthase improves memory and reduces amyloid-beta plaque load in transgenic mice.

Bernardo A, Harrison FE, McCord M, Zhao J, Bruchey A, Davies SS, Jackson Roberts L, Mathews PM, Matsuoka Y, Ariga T, Yu RK, Thompson R, McDonald MP
Neurobiol Aging. 2009 30 (11): 1777-91

PMID: 18258340 · DOI:10.1016/j.neurobiolaging.2007.12.022

MeSH Terms (22)

Alzheimer Disease Amyloid Amyloid beta-Protein Precursor Animals CD11b Antigen Cells, Cultured Cerebral Cortex Disease Models, Animal Gas Chromatography-Mass Spectrometry Humans Lipid Peroxidation Maze Learning Memory Memory Disorders Mice Mice, Transgenic Mutation Neurons Plaque, Amyloid Presenilin-1 Protein Binding Sialyltransferases

Connections (3)

This publication is referenced by other Labnodes entities: