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The publication data currently available has been vetted by Vanderbilt faculty, staff, administrators and trainees. The data itself is retrieved directly from NCBI's PubMed and is automatically updated on a weekly basis to ensure accuracy and completeness.

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Results: 1 to 5 of 5

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High Frequency of Ovarian Cyst Development in Vhl;Snf5 Mice.
Kuwahara Y, Kennedy LM, Karnezis AN, Mora-Blanco EL, Rogers AB, Fletcher CD, Huntsman DG, Roberts CWM, Rathmell WK, Weissman BE
(2018) Am J Pathol 188: 1510-1516
MeSH Terms: Animals, Female, Hemorrhage, Mice, Mice, Knockout, Mutation, Ovarian Cysts, Phenotype, SMARCB1 Protein, Von Hippel-Lindau Tumor Suppressor Protein
Added October 30, 2019
0 Communities
1 Members
0 Resources
MeSH Terms
SMARCB1 expression in epithelioid sarcoma is regulated by miR-206, miR-381, and miR-671-5p on Both mRNA and protein levels.
Papp G, Krausz T, Stricker TP, Szendrői M, Sápi Z
(2014) Genes Chromosomes Cancer 53: 168-76
MeSH Terms: Chromosomal Proteins, Non-Histone, DNA-Binding Proteins, Gene Expression Regulation, Neoplastic, Humans, MicroRNAs, RNA Interference, RNA, Messenger, SMARCB1 Protein, Sarcoma, Transcription Factors
Added March 28, 2014
0 Communities
1 Members
0 Resources
10 MeSH Terms
INI1/hSNF5-interaction defective HIV-1 IN mutants exhibit impaired particle morphology, reverse transcription and integration in vivo.
Mathew S, Nguyen M, Wu X, Pal A, Shah VB, Prasad VR, Aiken C, Kalpana GV
(2013) Retrovirology 10: 66
MeSH Terms: Cell Line, Chromosomal Proteins, Non-Histone, DNA-Binding Proteins, HIV Integrase, HIV-1, Host-Pathogen Interactions, Humans, Microscopy, Electron, Transmission, Reverse Transcription, SMARCB1 Protein, Transcription Factors, Virion, Virus Assembly, Virus Integration
Added May 27, 2014
0 Communities
1 Members
0 Resources
14 MeSH Terms
SMARCB1 protein and mRNA loss is not caused by promoter and histone hypermethylation in epithelioid sarcoma.
Papp G, Changchien YC, Péterfia B, Pecsenka L, Krausz T, Stricker TP, Khoor A, Donner L, Sápi Z
(2013) Mod Pathol 26: 393-403
MeSH Terms: Adolescent, Adult, Aged, Aged, 80 and over, Child, Chromosomal Proteins, Non-Histone, DNA Methylation, DNA Mutational Analysis, DNA-Binding Proteins, Down-Regulation, Enhancer of Zeste Homolog 2 Protein, Female, Gene Deletion, Gene Expression Regulation, Neoplastic, Genetic Predisposition to Disease, Histones, Humans, Immunohistochemistry, In Situ Hybridization, Fluorescence, Laser Capture Microdissection, Male, Middle Aged, Mutation, Phenotype, Poland, Polycomb Repressive Complex 2, Promoter Regions, Genetic, RNA, Messenger, Real-Time Polymerase Chain Reaction, Retrospective Studies, Reverse Transcriptase Polymerase Chain Reaction, SMARCB1 Protein, Sarcoma, Soft Tissue Neoplasms, Transcription Factors, United States, Young Adult
Added March 28, 2014
0 Communities
1 Members
0 Resources
37 MeSH Terms
Loss of SMARCB1/INI1 expression in poorly differentiated chordomas.
Mobley BC, McKenney JK, Bangs CD, Callahan K, Yeom KW, Schneppenheim R, Hayden MG, Cherry AM, Gokden M, Edwards MS, Fisher PG, Vogel H
(2010) Acta Neuropathol 120: 745-53
MeSH Terms: Cell Differentiation, Child, Child, Preschool, Chordoma, Chromosomal Proteins, Non-Histone, DNA-Binding Proteins, Female, Gene Deletion, Humans, Infant, Male, Mutation, Neoplasm Invasiveness, SMARCB1 Protein, Spinal Neoplasms, Transcription Factors
Added August 14, 2014
0 Communities
1 Members
0 Resources
16 MeSH Terms