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The publication data currently available has been vetted by Vanderbilt faculty, staff, administrators and trainees. The data itself is retrieved directly from NCBI's PubMed and is automatically updated on a weekly basis to ensure accuracy and completeness.

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Results: 1 to 10 of 104

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Differential Cell Susceptibilities to Kras in the Setting of Obstructive Chronic Pancreatitis.
Shi C, Pan FC, Kim JN, Washington MK, Padmanabhan C, Meyer CT, Kopp JL, Sander M, Gannon M, Beauchamp RD, Wright CV, Means AL
(2019) Cell Mol Gastroenterol Hepatol 8: 579-594
MeSH Terms: Acinar Cells, Animals, Carcinogenesis, Carcinoma, Pancreatic Ductal, Cell Transformation, Neoplastic, Disease Models, Animal, Genes, ras, Metaplasia, Mice, Mutation, Pancreatic Neoplasms, Pancreatitis, Chronic, Precancerous Conditions, Proto-Oncogene Proteins p21(ras), Signal Transduction
Added August 6, 2019
1 Communities
1 Members
0 Resources
15 MeSH Terms
Myeloid-derived interleukin-1β drives oncogenic KRAS-NF-κΒ addiction in malignant pleural effusion.
Marazioti A, Lilis I, Vreka M, Apostolopoulou H, Kalogeropoulou A, Giopanou I, Giotopoulou GA, Krontira AC, Iliopoulou M, Kanellakis NI, Agalioti T, Giannou AD, Jones-Paris C, Iwakura Y, Kardamakis D, Blackwell TS, Taraviras S, Spella M, Stathopoulos GT
(2018) Nat Commun 9: 672
MeSH Terms: Animals, Cell Line, Tumor, Chemokine CXCL1, Female, Genes, ras, Humans, I-kappa B Kinase, Interleukin-1beta, Male, Mice, Mice, Inbred C57BL, Mutation, Myeloid Cells, NF-kappa B, Pleural Effusion, Malignant, Receptors, Interleukin-1
Added March 21, 2018
0 Communities
1 Members
0 Resources
16 MeSH Terms
Molecular dissection of effector mechanisms of RAS-mediated resistance to anti-EGFR antibody therapy.
Kasper S, Reis H, Ziegler S, Nothdurft S, Mueller A, Goetz M, Wiesweg M, Phasue J, Ting S, Wieczorek S, Even A, Worm K, Pogorzelski M, Breitenbuecher S, Meiler J, Paul A, Trarbach T, Schmid KW, Breitenbuecher F, Schuler M
(2017) Oncotarget 8: 45898-45917
MeSH Terms: Antineoplastic Agents, Immunological, Apoptosis, Biomarkers, Cell Line, Tumor, Colorectal Neoplasms, Drug Resistance, Neoplasm, ErbB Receptors, Exons, Genes, ras, Humans, Mitogen-Activated Protein Kinases, Mutation, Odds Ratio, Phosphatidylinositol 3-Kinases, Proto-Oncogene Proteins c-akt, Proto-Oncogene Proteins c-bcl-2, Signal Transduction
Added March 17, 2018
0 Communities
1 Members
0 Resources
17 MeSH Terms
Mutational landscape of EGFR-, MYC-, and Kras-driven genetically engineered mouse models of lung adenocarcinoma.
McFadden DG, Politi K, Bhutkar A, Chen FK, Song X, Pirun M, Santiago PM, Kim-Kiselak C, Platt JT, Lee E, Hodges E, Rosebrock AP, Bronson RT, Socci ND, Hannon GJ, Jacks T, Varmus H
(2016) Proc Natl Acad Sci U S A 113: E6409-E6417
MeSH Terms: Adenocarcinoma, Adenocarcinoma of Lung, Animals, Carcinogens, Cell Transformation, Neoplastic, DNA Copy Number Variations, DNA Mutational Analysis, Disease Models, Animal, ErbB Receptors, Gene Dosage, Genes, myc, Genes, ras, Genome-Wide Association Study, Lung Neoplasms, Mice, Mice, Transgenic, Mutation, Point Mutation, Proto-Oncogene Mas, ROC Curve, Whole Exome Sequencing
Added April 26, 2017
0 Communities
1 Members
0 Resources
21 MeSH Terms
Expression of Activated Ras in Gastric Chief Cells of Mice Leads to the Full Spectrum of Metaplastic Lineage Transitions.
Choi E, Hendley AM, Bailey JM, Leach SD, Goldenring JR
(2016) Gastroenterology 150: 918-30.e13
MeSH Terms: Animals, Anticarcinogenic Agents, Benzimidazoles, Cell Differentiation, Cell Lineage, Cell Proliferation, Cell Transformation, Neoplastic, Chief Cells, Gastric, Disease Progression, Female, Gene Expression Regulation, Neoplastic, Genes, ras, Genetic Predisposition to Disease, Humans, Macrophages, Male, Metaplasia, Mice, Inbred C57BL, Mice, Transgenic, Mitogen-Activated Protein Kinase Kinases, Mutation, Phenotype, Protein Kinase Inhibitors, Signal Transduction, Stomach Neoplasms, Time Factors, Transcriptional Activation
Added March 28, 2016
0 Communities
2 Members
0 Resources
27 MeSH Terms
KRAS Genomic Status Predicts the Sensitivity of Ovarian Cancer Cells to Decitabine.
Stewart ML, Tamayo P, Wilson AJ, Wang S, Chang YM, Kim JW, Khabele D, Shamji AF, Schreiber SL
(2015) Cancer Res 75: 2897-906
MeSH Terms: Animals, Azacitidine, Biomarkers, Tumor, Cystadenocarcinoma, Serous, Decitabine, Drug Resistance, Neoplasm, Female, Genes, ras, Humans, Mice, Mice, Nude, Mice, Transgenic, Mutation, Ovarian Neoplasms, Prognosis, Tumor Cells, Cultured, Xenograft Model Antitumor Assays
Added February 22, 2016
0 Communities
1 Members
0 Resources
17 MeSH Terms
Ovarian mucinous tumor with malignant mural nodules: dedifferentiation or collision?
Desouki MM, Khabele D, Crispens MA, Fadare O
(2015) Int J Gynecol Pathol 34: 19-24
MeSH Terms: Carcinoma, Cell Movement, Cell Transformation, Neoplastic, Combined Modality Therapy, Drug Therapy, Female, Genes, ras, Humans, Mutation, Neoplasms, Cystic, Mucinous, and Serous, Ovarian Neoplasms, Ovariectomy, Proto-Oncogene Proteins B-raf, Treatment Outcome, Young Adult
Added February 19, 2015
0 Communities
1 Members
0 Resources
15 MeSH Terms
Beyond exon 2--the developing story of RAS mutations in colorectal cancer.
Berlin J
(2013) N Engl J Med 369: 1059-60
MeSH Terms: Antibodies, Monoclonal, Antineoplastic Combined Chemotherapy Protocols, Colorectal Neoplasms, ErbB Receptors, Fluorouracil, Genes, ras, Humans, Leucovorin, Organoplatinum Compounds, Panitumumab, Proto-Oncogene Proteins, Proto-Oncogene Proteins B-raf, Proto-Oncogene Proteins p21(ras), ras Proteins
Added March 20, 2014
0 Communities
1 Members
0 Resources
14 MeSH Terms
Distinct molecular features of colorectal cancer in Ghana.
Raskin L, Dakubo JC, Palaski N, Greenson JK, Gruber SB
(2013) Cancer Epidemiol 37: 556-61
MeSH Terms: African Americans, Colorectal Neoplasms, DNA Mismatch Repair, DNA, Neoplasm, Exons, Female, Genes, ras, Ghana, Humans, Immunohistochemistry, Male, Microsatellite Instability, Middle Aged, Molecular Epidemiology, Paraffin Embedding, Proto-Oncogene Proteins B-raf
Added March 7, 2014
0 Communities
1 Members
0 Resources
16 MeSH Terms
Identification of Sox9-dependent acinar-to-ductal reprogramming as the principal mechanism for initiation of pancreatic ductal adenocarcinoma.
Kopp JL, von Figura G, Mayes E, Liu FF, Dubois CL, Morris JP, Pan FC, Akiyama H, Wright CV, Jensen K, Hebrok M, Sander M
(2012) Cancer Cell 22: 737-50
MeSH Terms: Acinar Cells, Carcinoma, Pancreatic Ductal, Genes, ras, Humans, Metaplasia, Mutation, Pancreatic Neoplasms, Precancerous Conditions, SOX9 Transcription Factor
Added March 20, 2014
2 Communities
1 Members
0 Resources
9 MeSH Terms