Central sprouting and functional plasticity of regenerated primary afferents. Koerber HR, Mirnics K, Brown PB, Mendell LM (1994) J Neurosci 14: 3655-71 Release of carbon disulfide is a contributing mechanism in the axonopathy produced by N,N-diethyldithiocarbamate. Johnson DJ, Graham DG, Amarnath V, Amarnath K, Valentine WM (1998) Toxicol Appl Pharmacol 148: 288-96 Copper accumulation and lipid oxidation precede inflammation and myelin lesions in N,N-diethyldithiocarbamate peripheral myelinopathy. Viquez OM, Valentine HL, Amarnath K, Milatovic D, Valentine WM (2008) Toxicol Appl Pharmacol 229: 77-85 Plasticity of dorsal horn cell receptive fields after peripheral nerve regeneration. Koerber HR, Mirnics K (1996) J Neurophysiol 75: 2255-67 Properties of regenerated primary afferents and their functional connections. Koerber HR, Mirnics K, Mendell LM (1995) J Neurophysiol 73: 693-702
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