Pathogenesis of diquat-induced liver necrosis in selenium-deficient rats: assessment of the roles of lipid peroxidation and selenoprotein P. Burk RF, Hill KE, Awad JA, Morrow JD, Kato T, Cockell KA, Lyons PR (1995) Hepatology 21: 561-9 A series of prostaglandin F2-like compounds are produced in vivo in humans by a non-cyclooxygenase, free radical-catalyzed mechanism. Morrow JD, Hill KE, Burk RF, Nammour TM, Badr KF, Roberts LJ (1990) Proc Natl Acad Sci U S A 87: 9383-7 Transgenic mice overexpressing glutathione peroxidase 4 are protected against oxidative stress-induced apoptosis. Ran Q, Liang H, Gu M, Qi W, Walter CA, Roberts LJ, Herman B, Richardson A, Van Remmen H (2004) J Biol Chem 279: 55137-46 Centrilobular endothelial cell injury by diquat in the selenium-deficient rat liver. Atkinson JB, Hill KE, Burk RF (2001) Lab Invest 81: 193-200 Effect of selenium deficiency and glutathione-modulating agents on diquat toxicity and lipid peroxidation in rats. Awad JA, Burk RF, Roberts LJ (1994) J Pharmacol Exp Ther 270: 858-64
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