Pathogen-host interactions in Pseudomonas aeruginosa pneumonia. Sadikot RT, Blackwell TS, Christman JW, Prince AS (2005) Am J Respir Crit Care Med 171: 1209-23 The innate immune protein calprotectin promotes Pseudomonas aeruginosa and Staphylococcus aureus interaction. Wakeman CA, Moore JL, Noto MJ, Zhang Y, Singleton MD, Prentice BM, Gilston BA, Doster RS, Gaddy JA, Chazin WJ, Caprioli RM, Skaar EP (2016) Nat Commun 7: 11951 The Cytokine Response to Lipopolysaccharide Does Not Predict the Host Response to Infection. Fensterheim BA, Guo Y, Sherwood ER, Bohannon JK (2017) J Immunol 198: 3264-3273 Targeted immunomodulation of the NF-kappaB pathway in airway epithelium impacts host defense against Pseudomonas aeruginosa. Sadikot RT, Zeng H, Joo M, Everhart MB, Sherrill TP, Li B, Cheng DS, Yull FE, Christman JW, Blackwell TS (2006) J Immunol 176: 4923-30 Prostaglandin E2-induced changes in alveolar macrophage scavenger receptor profiles differentially alter phagocytosis of Pseudomonas aeruginosa and Staphylococcus aureus post-bone marrow transplant. Domingo-Gonzalez R, Katz S, Serezani CH, Moore TA, Levine AM, Moore BB (2013) J Immunol 190: 5809-17 Induction and function of lipocalin prostaglandin D synthase in host immunity. Joo M, Kwon M, Sadikot RT, Kingsley PJ, Marnett LJ, Blackwell TS, Peebles RS, Urade Y, Christman JW (2007) J Immunol 179: 2565-75 Prophylactic treatment with fms-like tyrosine kinase-3 ligand after burn injury enhances global immune responses to infection. Bohannon J, Cui W, Cox R, Przkora R, Sherwood E, Toliver-Kinsky T (2008) J Immunol 180: 3038-48 p47phox deficiency impairs NF-kappa B activation and host defense in Pseudomonas pneumonia. Sadikot RT, Zeng H, Yull FE, Li B, Cheng DS, Kernodle DS, Jansen ED, Contag CH, Segal BH, Holland SM, Blackwell TS, Christman JW (2004) J Immunol 172: 1801-8 Dendritic cell modification of neutrophil responses to infection after burn injury. Bohannon J, Cui W, Sherwood E, Toliver-Kinsky T (2010) J Immunol 185: 2847-53 Enhancement of dendritic cell production by fms-like tyrosine kinase-3 ligand increases the resistance of mice to a burn wound infection. Toliver-Kinsky TE, Cui W, Murphey ED, Lin C, Sherwood ER (2005) J Immunol 174: 404-10 A 14-year-old boy with kidney allograft failure in the first month after transplantation. Revelo MP, Paueksakon P, Goral S, Helderman H, Fogo AB (2000) Am J Kidney Dis 36: 871-4 Pseudomonas peritonitis in peritoneal dialysis patients: the Network #9 Peritonitis Study. Bunke M, Brier ME, Golper TA (1995) Am J Kidney Dis 25: 769-74 Bacterial clearance of Pseudomonas aeruginosa is enhanced by the inhibition of COX-2. Sadikot RT, Zeng H, Azim AC, Joo M, Dey SK, Breyer RM, Peebles RS, Blackwell TS, Christman JW (2007) Eur J Immunol 37: 1001-9 Role of G-CSF in monophosphoryl lipid A-mediated augmentation of neutrophil functions after burn injury. Bohannon JK, Luan L, Hernandez A, Afzal A, Guo Y, Patil NK, Fensterheim B, Sherwood ER (2016) J Leukoc Biol 99: 629-40 Role of granulocyte macrophage colony-stimulating factor during gram-negative lung infection with Pseudomonas aeruginosa. Ballinger MN, Paine R, Serezani CH, Aronoff DM, Choi ES, Standiford TJ, Toews GB, Moore BB (2006) Am J Respir Cell Mol Biol 34: 766-74 The capability of Pseudomonas aeruginosa to recruit zinc under conditions of limited metal availability is affected by inactivation of the ZnuABC transporter. D'Orazio M, Mastropasqua MC, Cerasi M, Pacello F, Consalvo A, Chirullo B, Mortensen B, Skaar EP, Ciavardelli D, Pasquali P, Battistoni A (2015) Metallomics 7: 1023-35 Gamma interferon does not enhance clearance of Pseudomonas aeruginosa but does amplify a proinflammatory response in a murine model of postseptic immunosuppression. Murphey ED, Herndon DN, Sherwood ER (2004) Infect Immun 72: 6892-901 The Toll-like receptor 4 agonist monophosphoryl lipid a augments innate host resistance to systemic bacterial infection. Romero CD, Varma TK, Hobbs JB, Reyes A, Driver B, Sherwood ER (2011) Infect Immun 79: 3576-87 IL-15 Superagonist Expands mCD8+ T, NK and NKT Cells after Burn Injury but Fails to Improve Outcome during Burn Wound Infection. Patil NK, Luan L, Bohannon JK, Guo Y, Hernandez A, Fensterheim B, Sherwood ER (2016) PLoS One 11: e0148452 Transfusion medicine illustrated. Profound piperacillin-mediated drug-induced immune hemolysis in a patient with cystic fibrosis. Gehrie E, Neff AT, Ciombor KK, Harris N, Seegmiller AC, Young PP (2012) Transfusion 52: 4-5 Conditional regulation of cyclooxygenase-2 in tracheobronchial epithelial cells modulates pulmonary immunity. Park GY, Hu N, Wang X, Sadikot RT, Yull FE, Joo M, Peebles RS, Blackwell TS, Christman JW (2007) Clin Exp Immunol 150: 245-54 The nuclear factor kappa-B pathway in airway epithelium regulates neutrophil recruitment and host defence following Pseudomonas aeruginosa infection. Chen SM, Cheng DS, Williams BJ, Sherrill TP, Han W, Chont M, Saint-Jean L, Christman JW, Sadikot RT, Yull FE, Blackwell TS (2008) Clin Exp Immunol 153: 420-8 Pretreatment with the Gram-positive bacterial cell wall molecule peptidoglycan improves bacterial clearance and decreases inflammation and mortality in mice challenged with Pseudomonas aeruginosa. Murphey ED, Sherwood ER (2008) Microbes Infect 10: 1244-50 Pseudomonas aeruginosa: host defence in lung diseases. Williams BJ, Dehnbostel J, Blackwell TS (2010) Respirology 15: 1037-56 Glucan phosphate treatment attenuates burn-induced inflammation and improves resistance to Pseudomonas aeruginosa burn wound infection. Lyuksutova OI, Murphey ED, Toliver-Kinsky TE, Lin CY, Cui W, Williams DL, Sherwood ER (2005) Shock 23: 224-32 Bacterial clearance and mortality are not improved by a combination of IL-10 neutralization and IFN-gamma administration in a murine model of post-CLP immunosuppression. Murphey ED, Sherwood ER (2006) Shock 26: 417-24 Improved bacterial clearance and decreased mortality can be induced by LPS tolerance and is not dependent upon IFN-gamma. Murphey ED, Fang G, Varma TK, Sherwood ER (2007) Shock 27: 289-95 Fms-like tyrosine kinase-3 ligand alters antigen-specific responses to infections after severe burn injury. Bohannon J, Fang G, Cui W, Sherwood E, Toliver-Kinsky T (2009) Shock 32: 435-41 Fms-like tyrosine kinase-3 ligand increases resistance to burn wound infection through effects on plasmacytoid dendritic cells. Bae L, Bohannon JK, Cui W, Vinish M, Toliver-Kinsky T (2017) BMC Immunol 18: 9 Endogenous Fms-like tyrosine kinase-3 ligand levels are not altered in mice after a severe burn and infection. Bohannon JK, Cui W, Toliver-Kinsky T (2009) BMC Immunol 10: 47 Effect of transforming growth factor-beta neutralization on survival and bacterial clearance in a murine model of Pseudomonas aeruginosa burn wound infection. Huang Z, Pereira C, Toliver-Kinsky T, Murphey ED, Varma TK, Lin CY, Herndon DN, Sherwood ER (2006) J Burn Care Res 27: 682-7
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