Origin of replication of the DNA of a herpesvirus (pseudorabies). Ben-Porat T, Veach RA (1980) Proc Natl Acad Sci U S A 77: 172-5 Recombination occurs mainly between parental genomes and precedes DNA replication in pseudorabies virus-infected cells. Ben-Porat T, Brown L, Veach RA (1982) J Virol 44: 134-43 Acquisition of an additional internal cleavage site differentially affects the ability of pseudorabies virus to multiply in different host cells. Rall GF, Lu ZQ, Sugg N, Veach RA, Ben-Porat T (1991) J Virol 65: 6604-11 Proteins specified by the short unique region of the genome of pseudorabies virus play a role in the release of virions from certain cells. Ben-Porat T, DeMarchi J, Pendrys J, Veach RA, Kaplan AS (1986) J Virol 57: 191-6 Functions of the sequences at the ends of the inverted repeats of pseudorabies virus. Rall GF, Kupershmidt S, Sugg N, Veach RA, Ben-Porat T (1992) J Virol 66: 1506-19 Role of the central melanocortin circuitry in adaptive thermogenesis of brown adipose tissue. Voss-Andreae A, Murphy JG, Ellacott KL, Stuart RC, Nillni EA, Cone RD, Fan W (2007) Endocrinology 148: 1550-60 Functions of the major nonstructural DNA binding protein of a herpesvirus (pseudorabies). Ben-Porat T, Veach RA, Hampl H (1983) Virology 124: 411-24 Localization of the regions of homology between the genomes of herpes simplex virus, type 1, and pseudorabies virus. Ben-Porat T, Veach RA, Ihara S (1983) Virology 127: 194-204 Replication of herpesvirus DNA. VI. Virions containing either isomer of pseudorabies virus DNA are infectious. Ben-Porat T, Veach RA, Ladin BF (1980) Virology 102: 370-80 Equalization of the inverted repeat sequences of the pseudorabies virus genome by intermolecular recombination. Ben-Porat T, Deatly A, Veach RA, Blankenship ML (1984) Virology 132: 303-14 Differential association with cellular substructures of pseudorabies virus DNA during early and late phases of replication. Ben-Porat T, Veach RA, Blankenship ML, Kaplan AS (1984) Virology 139: 205-22
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