1. Transcriptional regulation by the t(8;21) fusion protein, AML-1/ETO. Hiebert SW, Downing JR, Lenny N, Meyers S (1996) Curr Top Microbiol Immunol : 253-8
    › Primary publication · 8585956 (PubMed)
  2. The t(12;21) translocation converts AML-1B from an activator to a repressor of transcription. Hiebert SW, Sun W, Davis JN, Golub T, Shurtleff S, Buijs A, Downing JR, Grosveld G, Roussell MF, Gilliland DG, Lenny N, Meyers S (1996) Mol Cell Biol 16(4): 1349-55
    › Primary publication · 8657108 (PubMed) · PMC231119 (PubMed Central)
  3. Synergistic up-regulation of the myeloid-specific promoter for the macrophage colony-stimulating factor receptor by AML1 and the t(8;21) fusion protein may contribute to leukemogenesis. Rhoades KL, Hetherington CJ, Rowley JD, Hiebert SW, Nucifora G, Tenen DG, Zhang DE (1996) Proc Natl Acad Sci U S A 93(21): 11895-900
    › Primary publication · 8876234 (PubMed) · PMC38155 (PubMed Central)
  4. Rearrangement of the AML1/CBFA2 gene in myeloid leukemia with the 3;21 translocation: expression of co-existing multiple chimeric genes with similar functions as transcriptional repressors, but with opposite tumorigenic properties. Zent C, Kim N, Hiebert S, Zhang DE, Tenen DG, Rowley JD, Nucifora G (1996) Curr Top Microbiol Immunol : 243-52
    › Primary publication · 8585955 (PubMed)
  5. Prostaglandin A2 blocks the activation of G1 phase cyclin-dependent kinase without altering mitogen-activated protein kinase stimulation. Hitomi M, Shu J, Strom D, Hiebert SW, Harter ML, Stacey DW (1996) J Biol Chem 271(16): 9376-83
    › Primary publication · 8621603 (PubMed)
  6. CCAAT enhancer-binding protein (C/EBP) and AML1 (CBF alpha2) synergistically activate the macrophage colony-stimulating factor receptor promoter. Zhang DE, Hetherington CJ, Meyers S, Rhoades KL, Larson CJ, Chen HM, Hiebert SW, Tenen DG (1996) Mol Cell Biol 16(3): 1231-40
    › Primary publication · 8622667 (PubMed) · PMC231105 (PubMed Central)
  7. An AML-1 consensus sequence binds an osteoblast-specific complex and transcriptionally activates the osteocalcin gene. Banerjee C, Hiebert SW, Stein JL, Lian JB, Stein GS (1996) Proc Natl Acad Sci U S A 93(10): 4968-73
    › Primary publication · 8643513 (PubMed) · PMC39389 (PubMed Central)
  8. AML-2 is a potential target for transcriptional regulation by the t(8;21) and t(12;21) fusion proteins in acute leukemia. Meyers S, Lenny N, Sun W, Hiebert SW (1996) Oncogene 13(2): 303-12
    › Primary publication · 8710369 (PubMed)
  9. AML1, the target of multiple chromosomal translocations in human leukemia, is essential for normal fetal liver hematopoiesis. Okuda T, van Deursen J, Hiebert SW, Grosveld G, Downing JR (1996) Cell 84(2): 321-30
    › Primary publication · 8565077 (PubMed)
  10. The tissue-specific nuclear matrix protein, NMP-2, is a member of the AML/CBF/PEBP2/runt domain transcription factor family: interactions with the osteocalcin gene promoter. Merriman HL, van Wijnen AJ, Hiebert S, Bidwell JP, Fey E, Lian J, Stein J, Stein GS (1995) Biochemistry 34(40): 13125-32
    › Primary publication · 7548073 (PubMed)