Nathan Mundell
Last active: 3/20/2012


Nathan is a graduate student in the Labosky lab and joined the department of Pharmacology after beginning the Interdisciplinary Graduate Program in Biomedical Sciences in 2004. Previously Nathan graduated from Western Kentucky University with a BS in Biology in 2002 and then worked on the role of transforming growth factor beta in heart development as a Research Assistant at Vanderbilt with Dr. Joey Barnett. His project in the Labosky lab focuses on understanding the molecular regulation of self-renewal and multipotency of neural crest stem cells. More specifically, he is using a Wnt1Cre transgenic allele to drive neural crest-specific deletion of the transcription factor Foxd3 in mice. By in vitro culture of neural crest progenitors from Foxd3 mutant embryos, it is possible to dissect the requirement of this gene for maintenance of multipotent neural crest subpopulations. He is currently funded by a predoctoral Ruth L. Kirschstein NRSA fellowship from the National Institutes of Neurological Disorders and Stroke.


The following timeline graph is generated from all co-authored publications.

Most recent publication(s) are shown below:

  1. Requirement for Foxd3 in the maintenance of neural crest progenitors. Teng L, Mundell NA, Frist AY, Wang Q, Labosky PA (2008) Development 135(9): 1615-24
    › Primary publication · 18367558 (PubMed) · PMC2562748 (PubMed Central)
  2. Efficient replication, and evolution of Sindbis virus genomes with non-canonical 3'A/U-rich elements (NC3ARE) in neonatal mice. James FD, Hietala KA, Eldar D, Guess TE, Cone C, Mundell NA, Mundall N, Barnett JV, Raju R (2007) Virus Genes 35(3): 651-62
    › Primary publication · 17616797 (PubMed)
  3. Transforming growth factor-beta induces loss of epithelial character and smooth muscle cell differentiation in epicardial cells. Compton LA, Potash DA, Mundell NA, Barnett JV (2006) Dev Dyn 235(1): 82-93
    › Primary publication · 16258965 (PubMed)
  4. Transforming growth factor-beta stimulates epithelial-mesenchymal transformation in the proepicardium. Olivey HE, Mundell NA, Austin AF, Barnett JV (2006) Dev Dyn 235(1): 50-9
    › Primary publication · 16245329 (PubMed) · PMC3160345 (PubMed Central)
  5. Activin receptor-like kinase 2 and Smad6 regulate epithelial-mesenchymal transformation during cardiac valve formation. Desgrosellier JS, Mundell NA, McDonnell MA, Moses HL, Barnett JV (2005) Dev Biol 280(1): 201-10
    › Primary publication · 15766759 (PubMed)