SYSTEM MESSAGE: Be advised of scheduled maintenace on July 30, 2014 starting at 9AM CST during which this site will not be available.
 
The Vanderbilt Diabetes Research and Training Center (DRTC) is a NIH-sponsored Diabetes Center that facilitates the discovery, application, and translation of scientific knowledge to improve the care of patients with diabetes.
 

The Vanderbilt DRTC, an interdisciplinary program involving more than 120 faculty distributed among 15 departments in 3 schools and 4 colleges at Vanderbilt and neighboring Meharry Medical College, consists of:

  • Administrative Component
  • Enrichment, Training, and Outreach Program that fosters an environment conducive to collaborative, interdisciplinary research (DRTC Seminar SeriesVanderbilt Diabetes Day), and to training new diabetes scientists.

Publications/Citations

Most recent publications

  1. Protease-activated receptor (PAR) 1 and PAR4 differentially regulate factor V expression from human platelets. Duvernay M, Young S, Gailani D, Schoenecker J, Hamm HE, Hamm H (2013) Mol Pharmacol 83(4): 781-92
    › Primary publication · 23307185 (PubMed) · PMC3608438 (PubMed Central)
  2. The relationship of oxidative stress, adiposity and metabolic risk factors in healthy Black and White American youth. Warolin J, Coenen KR, Kantor JL, Whitaker LE, Wang L, Acra SA, Roberts LJ, Buchowski MS (2014) Pediatr Obes 9(1): 43-52
    › Primary publication · 23296459 (PubMed) · PMC3775931 (PubMed Central)
  3. The best-laid plans. Harris RC (2013) Am J Physiol Renal Physiol 304(8): F1086-7
    › Primary publication · 23269642 (PubMed) · PMC3625842 (PubMed Central)
  4. Inflammation during obesity is not all bad: evidence from animal and human studies. Ye J, McGuinness OP (2013) Am J Physiol Endocrinol Metab 304(5): E466-77
    › Primary publication · 23269411 (PubMed) · PMC3774179 (PubMed Central)
  5. The yeast eukaryotic translation initiation factor 2B translation initiation complex interacts with the fatty acid synthesis enzyme YBR159W and endoplasmic reticulum membranes. Browne CM, Samir P, Fites JS, Villarreal SA, Link AJ (2013) Mol Cell Biol 33(5): 1041-56
    › Primary publication · 23263984 (PubMed) · PMC3623073 (PubMed Central)
  6. Neuropeptide Y and somatostatin inhibit insulin secretion through different mechanisms. Schwetz TA, Ustione A, Piston DW (2013) Am J Physiol Endocrinol Metab 304(2): E211-21
    › Primary publication · 23211512 (PubMed) · PMC3543566 (PubMed Central)
  7. Islet α-, β-, and δ-cell development is controlled by the Ldb1 coregulator, acting primarily with the islet-1 transcription factor. Hunter CS, Dixit S, Cohen T, Ediger B, Wilcox C, Ferreira M, Westphal H, Stein R, May CL (2013) Diabetes 62(3): 875-86
    › Primary publication · 23193182 (PubMed) · PMC3581213 (PubMed Central)
  8. Molecular mechanisms and the role of saturated fatty acids in the progression of non-alcoholic fatty liver disease. Leamy AK, Egnatchik RA, Young JD (2013) Prog Lipid Res 52(1): 165-74
    › Primary publication · 23178552 (PubMed)
  9. Liver-specific disruption of the murine glucagon receptor produces α-cell hyperplasia: evidence for a circulating α-cell growth factor. Longuet C, Robledo AM, Dean ED, Dai C, Ali S, McGuinness I, de Chavez V, Vuguin PM, Charron MJ, Powers AC, Drucker DJ (2013) Diabetes 62(4): 1196-205
    › Primary publication · 23160527 (PubMed) · PMC3609565 (PubMed Central)
  10. 3-dimensional resin casting and imaging of mouse portal vein or intrahepatic bile duct system. Walter TJ, Sparks EE, Huppert SS (2012) J Vis Exp (68): e4272
    › Primary publication · 23128398 (PubMed)

Community Leaders

Contact Information

2215 Garland Ave.
807 Light Hall
Nashville, TN 37232-0202
USA
615-322-7990 (p)
615-343-0172 (f)

Terri Ray
615-936-7678 (p)
615-936-0063 (f)
Email

Keywords

type 1 diabetes (5), cores (2), NIDDK (1), type 2 diabetes (2), DRTC (3)